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Geriau pries nes pasisavinimas geresnis. Su visais vaistais taip, bet buna dirgina skrandy, jei pro valgio, poveikis buna prislopinamas

azuolyno bicas rašė:

Pries trefke, arba ryte!
Nebutina I vena, nes I vena rekomenduoja tik todel kad Kai kuriem suleidus I raumeny isberia

AZ- ka jis realiai duoda?

Mildronate [3-(2,2,2-trimethylhydrazinium)propionate dihydrate] ameliorates cardiac function during ischemia by modulating myocardial energy metabolism. Biochemical and pharmacological evidence supports the hypothesis that the mechanism of action of mildronate is based on its regulatory effect on carnitine concentration, whereby mildronate treatment shifts the myocardial energy metabolism from fatty acid oxidation to the more favorable glucose oxidation under ischemic conditions. Because mildronate treatment prepares cellular metabolism and membrane structures to survive ischemic stress conditions, it is possible that mildronate could be regarded as an agent of pharmacological preconditioning.

Mildronate (3-(2,2,2-trimethylhydrazinium)propionate; MET-88; meldonium, quaterine) is an antiischemic drug developed at the Latvian Institute of Organic Synthesis. Mildronate was designed to inhibit carnitine biosynthesis in order to prevent accumulation of cytotoxic intermediate products of fatty acid β-oxidation in ischemic tissues and to block this highly oxygen-consuming process. Mildronate is efficient in the treatment of heart ischemia and its consequences. Extensive evaluation of pharmacological activities of mildronate revealed its beneficial effect on cerebral circulation disorders and central nervous system (CNS) functions. The drug is used in neurological clinics for the treatment of brain circulation disorders. It appears to improve patients' mood; they become more active, their motor dysfunction decreases, and asthenia, dizziness and nausea become less pronounced. Since the brain does not utilize fatty acids as fuel other mechanisms of action of mildronate in CNS should be considered. Several reports indicate the possible existence of an alternative, non-carnitine dependent mechanism of action of mildronate. Our recent findings suggest that CNS effects of mildronate could be mediated by stimulation of the nitric oxide production in the vascular endothelium by modification of the γ-butyrobetaine and its esters pools. It is hypothesized that mildronate may increase the formation of the γ-butyrobetaine esters. The latter are potent cholinomimetics and may activate eNOS via acetylcholine receptors or specific γ-butyrobetaine ester receptors. This article summarizes known pharmacological effects of mildronate, its pharmacokinetics, toxicology, as well as the proposed mechanisms of action.

Man atrodo, dabar mildronatas šauna į top3 PED preparatus Jei taip reklamuoja reiks ir man nusipirkt jei dar vaistinėse liko

Jau uzsipirkta ir nesuprantu tiek metu buvo naudojamas, o db staiga pavirto i dopinga...

na realiai is jo naudos nera bet va kad visur ispirkta, ot padare reklamos sharapova, galetu nauja kontrakta pasirasyt su grindeks

hello123 rašė:

Sveiki , esu cia naujoke pradesiu gerti ReduxCut ,bet daug kur skaiciau kad permusineja labai sirdi nuo tu tableciu,ir galvoju gal gerti kokius vitaminus sirdziai pastiprinti ,arba MILDRONATE ar padetu?

Reduxcut yra amfos prikistas produktas. Sveiko proto zmogus jo nenaudos

hello123 rašė:

Gal tada zinot koki gera riebalu degintoja ?

Subalansuota mityba, sveikas gyvenimo būdas.

hello123 rašė:

Gal tada zinot koki gera riebalu degintoja ?

cleanbuterol

o gal yra bandziusiu ReduxCut?

Pastebejau kad po seto sarabanai sukas,ar yra kas galetu pagelbet tokiu atveju?